By S. Sarantseva
ISBN-10: 9533072539
ISBN-13: 9789533072531
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Extra info for Amyloidosis - Mechanisms and Prospects for Therapy
Example text
Circulation, Vol. 108, No. 24, (December 2003), pp. J. & Westermark, P. (2010). Amyloid fibril protein nomenclature: 2010 recommendations from the nomenclature committee of the International Society of Amyloidosis. 17, No. 3-4, (September 2010), pp. , Bergström, J. & Uchino, M. (2006). FK506 inhibits murine AA amyloidosis: possible involvement of T cells in amyloidogenesis. Journal of Rheumatology, Vol. 33, No. 11, (November 2006), pp. P. & Simon, A. (2008). 1 to degradation by MMP-1: potential explanation for higher risk of type AA amyloidosis.
Tuberculosis and leprosy are important causes of AA amyloidosis where these infections are endemic. Chronic osteomyelitis, bronchiectasis, chronically infected burns, and decubitus ulcers as well as the chronic pyelonephritis of paraplegic patients are other well-recognized associations. Castleman’s disease, Hodgkin’s lymphoma and renal carcinoma, which often cause fever, other systemic symptoms, and a major acute phase response, are the malignancies most commonly associated with systemic AA amyloidosis.
FK506 inhibits murine AA amyloidosis: possible involvement of T cells in amyloidogenesis. Journal of Rheumatology, Vol. 33, No. 11, (November 2006), pp. P. & Simon, A. (2008). 1 to degradation by MMP-1: potential explanation for higher risk of type AA amyloidosis. Rheumatology (Oxford), Vol. 47, No. 11, (November 2008), pp. W. & Simon, A. (2009). Cathepsin D activity protects against development of type AA amyloid fibrils. European Journal of Clinical Investigation, Vol. 39, No. 5, (May 2009), pp.
Amyloidosis - Mechanisms and Prospects for Therapy by S. Sarantseva
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